Here is another author advocating low starch diet for IBS. But as she says it also applies to IBD and AS (an autoimmune disease supposed to be linked to Crohn's). It is very similar to the SCD
http://www.everydiet.org/diet/low-starch-diet
Here is also some scientific data on link between AS, CD, and UC and how a low starch diet can help
http://www.kickas.org/as_dietary_primer.shtml
"The similarities between RA and the three conditions of AS, UC, and CD, would indicate that although there are different factors involved in the production of RA, a very similar overt expression in autoimmune terms does exist; joints become inflamed and might deteriorate, bone calcium management is affected, movements are painful, and many other ailments can appear. There is a genetic predisposition in both situations, and both are due to antigen—antibody involvement, which has been identified in both cases: The antigen (germ) reservoir in RA is likely the kidney-bladder system for proteus mirabilis, which clinically affects mostly persons with the HLA DR4 marker. The antigen reservoir for AS, UC, and CD is likely focii within the intestines for klebsiella pneumoniae, which affects persons with the HLA B27 marker and similar genotypes, as well as other individuals which may have uniquely higher susceptibility to K. pneumoniae infection (4).
Individuals with AS, CD, and UC all exhibit elevated numbers for klebsiella specific IgA, but test normal for proteus mirabilis specific IgG. Conversely, those who suffer with RA test normal for IgA, but have elevated proteus specific IgG. All subjects have permeable intestinal membranes most likely due to NSAID usage (2). Further, it has been proven that many other expected enterobacteriaceae are either effectively excluded by the bowel mucosa, or do not elicit any similar immune response."
"Reduced Starch Diet
It has been demonstrated that altering the normal diet to exclude foods which contain higher levels of starch, can significantly reduce the quantity of K. pneumoniae in the feces of all individuals (5). The implication is that the balance of intestinal florae can be altered to the extent that much fewer bacteria are available to cross the intestinal mucosa and elicit the production of IgA.
The low starch diet has been employed extensively in the treatment of AS at the AS Clinic located at Middlesex Hospital in London, under studies conducted by Dr Alan Ebringer (5). In these studies, significant reduction in IgA was obtained by reducing dietary starches by 40%, and a corresponding decrease in ESR was achieved by patients suffering from AS. This measure usually results in a considerable decrease of symptoms, typically unto effective remission. In patients unable to assiduously follow the low starch regimen, supplemental NSAID usage is required, however, most of the more recalcitrant cases could be treated using just the diet and the drug sulphasalazine. This has usually obviated the need for more powerful NSAIDs, avoiding their deleterious side effects (5)."
http://www.everydiet.org/diet/low-starch-diet
Here is also some scientific data on link between AS, CD, and UC and how a low starch diet can help
http://www.kickas.org/as_dietary_primer.shtml
"The similarities between RA and the three conditions of AS, UC, and CD, would indicate that although there are different factors involved in the production of RA, a very similar overt expression in autoimmune terms does exist; joints become inflamed and might deteriorate, bone calcium management is affected, movements are painful, and many other ailments can appear. There is a genetic predisposition in both situations, and both are due to antigen—antibody involvement, which has been identified in both cases: The antigen (germ) reservoir in RA is likely the kidney-bladder system for proteus mirabilis, which clinically affects mostly persons with the HLA DR4 marker. The antigen reservoir for AS, UC, and CD is likely focii within the intestines for klebsiella pneumoniae, which affects persons with the HLA B27 marker and similar genotypes, as well as other individuals which may have uniquely higher susceptibility to K. pneumoniae infection (4).
Individuals with AS, CD, and UC all exhibit elevated numbers for klebsiella specific IgA, but test normal for proteus mirabilis specific IgG. Conversely, those who suffer with RA test normal for IgA, but have elevated proteus specific IgG. All subjects have permeable intestinal membranes most likely due to NSAID usage (2). Further, it has been proven that many other expected enterobacteriaceae are either effectively excluded by the bowel mucosa, or do not elicit any similar immune response."
"Reduced Starch Diet
It has been demonstrated that altering the normal diet to exclude foods which contain higher levels of starch, can significantly reduce the quantity of K. pneumoniae in the feces of all individuals (5). The implication is that the balance of intestinal florae can be altered to the extent that much fewer bacteria are available to cross the intestinal mucosa and elicit the production of IgA.
The low starch diet has been employed extensively in the treatment of AS at the AS Clinic located at Middlesex Hospital in London, under studies conducted by Dr Alan Ebringer (5). In these studies, significant reduction in IgA was obtained by reducing dietary starches by 40%, and a corresponding decrease in ESR was achieved by patients suffering from AS. This measure usually results in a considerable decrease of symptoms, typically unto effective remission. In patients unable to assiduously follow the low starch regimen, supplemental NSAID usage is required, however, most of the more recalcitrant cases could be treated using just the diet and the drug sulphasalazine. This has usually obviated the need for more powerful NSAIDs, avoiding their deleterious side effects (5)."