ReeRee said:
I'll give it a shot. Can't hurt huh?
I am taking Methotrexate injections now and they cause some pretty wicked hot flashes. I'm taking Black Cohosh for it and it's working wonders. So a little ginseng couldn't hurt.
In some cases it can cause heartburn or dermatitis. Taking too much (I mean a hell of a lot) can cause CNS depression and/or arrythmia. It can also interact negatively with certain meds (antihypertensives, anticoagulants, antacid meds and , hypoglycemics) If it has enough activity to help then it has enough activity to possibly cause a possible adverse reaction. If there is no possibility of an adverse reaction, then it doesn't have enough biological activity to do anything at all.
People who are selling something (especially if they tout it as natural) and tell you that it will help you with no possibility of an adverse reaction are BSing you.
I have seen studies on the use of Giner to help with nausea and vomiting either post-op or associated with preganancy. I have seen a study involving the prevention of cancer formation in chronically inflamed area using ginseng.
abstract
http://jn.nutrition.org/cgi/content/abstract/137/1/183S
Inflammation, Cancer, and Targets of Ginseng1–3,
Lorne J. Hofseth4,* and Michael J. Wargovich5
4 Department of Basic Pharmaceutical Sciences, South Carolina College of Pharmacy and 5 Department of Pathology and Microbiology, School of Medicine, University of South Carolina, Columbia, SC 29208
* To whom correspondence should be addressed. E-mail:
hofseth@cop.sc.edu.
Chronic inflammation is associated with a high cancer risk. At the molecular level, free radicals and aldehydes, produced during chronic inflammation, can induce deleterious gene mutation and posttranslational modifications of key cancer-related proteins. Other products of inflammation, including cytokines, growth factors, and transcription factors such as nuclear factor B, control the expression of cancer genes (e.g., suppressor genes and oncogenes) and key inflammatory enzymes such as inducible nitric oxide synthase and cyclooxygenase-2. These enzymes in turn directly influence reactive oxygen species and eicosanoid levels. The procancerous outcome of chronic inflammation is increased DNA damage, increased DNA synthesis, cellular proliferation, disruption of DNA repair pathways and cellular milieu, inhibition of apoptosis, and promotion of angiogenesis and invasion. Chronic inflammation is also associated with immunosuppression, which is a risk factor for cancer. Current treatment strategies for reactive species overload diseases are frequently aimed at treating or preventing the cause of inflammation. Although these strategies have led to some progress in combating reactive species overload diseases and associated cancers, exposure often occurs again after eradication, treatment to eradicate the cause fails, or the treatment has long-term side effects. Therefore, the identification of molecules and pathways involved in chronic inflammation and cancer is critical to the design of agents that may help in preventing the progression of reactive species overload disease and cancer associated with disease progression. Here, we use ginseng as an example of an antiinflammatory molecule that targets many of the key players in the inflammation-to-cancer sequence.
